Wednesday 30th January - creatine & my training

This morning I weighed myself and I have put on another 1lb.  It doesn’t show itself to be water (which I thought it might be, due to the creatine), but I am taking ethyl esther which is a better creatine to take (see info below)

Creatine Ethyl Ester Info And Products
Absorbed Better Than Regular Creatine Monohydrate!

What is it and where does it come from?

Creatine Ethyl Ester HCL (CEE) is creatine monohydrate with an ester attached. Esters are organic compounds that are formed by esterification - the reaction of carboxylic acid and alcohols.

What does it do and what scientific studies give evidence to support this?
Regular creatine monohydrate has been shown effective at increasing lean muscle mass1,2,3,4, muscle strength5,6 and athletic performance.7,8However, regular creatine monohydrate is absorbed poorly by the body - and its effectiveness is dependant upon the cells ability to absorb it. The poor absorption rate of regular creatine monohydrate requires the creatine user to ingest large dosages of creatine to achieve desired effect.Because creatine draws water to the cell, and because most ingested creatine monohydrate is not absorbed, unabsorbed creatine will sit outside of the target cell with the water, and this will result in the “creatine bloat.”Long-term clinical studies have proven that creatine monohydrate is safe for use by persons free of medical complication9, but why would you want to ingest more creatine monohydrate than you have to simply because your creatine is inefficient?Creatine ethyl ester is creatine monohydrate with an ester attached. The attachment of an ester is significant, because esters are found in the fat tissue of animals. But, why is this important? What role does this have in the absorption of creatine?All substances that you put into your body will affect its operation. There are three ways that substances can affect a cells operation. They are:

  1. Ligand binding to protein receptor sites.
  2. Secondary messenger / metabotropic systems
  3. Passive permeation of the cell wall via lipids

When a substance enters the body and affects the bodies operation, it is known as a ligand. The soma and dendrites of the cell have protein receptor sites to which ligands can bind. The process of a ligand binding with a receptor site is akin to a lock and key: only keys of a certain shape work with certain locks. When they work and cause the cells stimulation they are called agonists. When they block the cell from functioning they are called antagonists.

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When a ligand binds with the receptor site of a target cell, the cell, in the simplest of cases, changes its shape, opens up its ion channels and changes its function. In so-called “secondary messenger” or metabotropic cells, the ligand binds with the receptor site and an internal protein known as a g-protein is released. This released protein then binds to an internal site inside of the cell, and then the cell changes its behavior by opening its ion channels. Cells that operate in this way are known as metabotropic cells because their operation requires metabolic energy.

Passive permeation is a process that describes the diffusion of a substance across a cell membrane through the use of lipids as transport mechanisms. Because no “work” is being done by the cell in this model, this model is called passive permeation.

Creatine monohydrate utilizes lipids to permeate the cell wall and enter the cell. Because of this, the esterification of creatine, and the presence of esters in animal fat tissue, becomes significant.

Creatine monohydrate is semi-lipopholic. This means that it inefficiently uses fat as a transport mechanism. The esterification of substances will increase their lipopholic abilities, and thus esterified creatine will use fat more efficiently to permeate the cell wall and exert its effects upon cellular function than its unesterified creatine monohydrate counterpart.

This means, simply, that not only will dosage requirements be lower, but the absorption of esterified creatine will be increased and the infamous “creatine bloat” will be eliminated!

But I feel I am still holding a little water, but not as much as I have done in the past by taking just creatine monohydrate. 

Yesterdays workout was hard, I could still feel the effects a little from Saturdays workout, and right now (Weds evening) I can still feel my lats from Sat too!

I decided to continue with the same shoulder and arms plan as last week, but to cut out the rest pause sets and maybe to do the longer reps to get a real pump in my shoulders.  Military press went well and I thought my lateral raises were going well too, that was until Simon corrected my technique again.  I thought I had mastered it from Saturdays workout (as I was turning out too much in my shoulders on the raises), but I still was a little and once corrected, I was on my way to getting a pump.  I also was able to get a spot on my dumbell shoulder presses with the 15kg.  I did the usual pressdowns and cable curl warmups for my arms  and then did incline supination curls, hammer curls for biceps and close grip push and press and………….my energy had dipped.  I left out the dips today and went onto chest.  I have to be very careful when getting tired training, you almost feel guilty for stopping, or just not doing one more set (exercise junkie!) but I am realising, that once you’re blown, you must stop or you just won’t grow and to tire out the central nervous system when you have a long day ahead is just not the way to go!  Especially when you want to win in 2008.

When I got home today the BNBF 2008 dvd has arrived, will watch that tomorrow.  Have read an interesting article on Jenny Garside.  She is a trained figure competitor that won the BNBF and NPA last year and just took first place at the Worlds in 2007.  She will be on the dvd, so looking forward to her standard of posing and watching her routine. 

Got to get ready, for its legs day tomorrow and clients.

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